About: Isabelle Plo

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An Entity of Type : foaf:Person, within Data Space : data.idref.fr associated with source document(s)

Attributes	Values
type	Person
sameAs	Plo, Isabelle (19..-....) Plo, Isabelle (19..-....)
alternative label	Azevedo, Isabelle Plo-Azevedo, Isabelle
Life Event	http://www.idref.fr/068888376/birth
preferred label	Plo, Isabelle (19..-....)
name	Isabelle Plo
dbpedia-owl:citizenship	http://sws.geonames.org/3017382/
Language	http://lexvo.org/id/iso639-3/fra
Given name	Isabelle
familyName	Plo
has biographical i...tion (Deprecated)	Chercheuse à l'Inserm, membre de l'U 1170 - Hématopoeïse normale et pathologique à l'Institut Gustave Roussy (en 2019) Titulaire d'un doctorat en physiopathologie humaine à Toulouse 3 en 2002
gender	female
ORCID iD	0000-0002-5915-6910
Scopus ID	6602730814
is sameAs of	Plo, Isabelle (19..-....) Plo, Isabelle (19..-....) Plo, Isabelle (19..-....)
is Thesis advisor of	http://www.sudoc.fr/191484245/id http://www.sudoc.fr/191936936/id Étude du rôle de HSF1 dans l'érythropoïèse http://www.sudoc.fr/257169237/id http://www.sudoc.fr/272310468/id http://www.sudoc.fr/234665920/id http://www.sudoc.fr/269738231/id http://www.sudoc.fr/233532358/id http://www.sudoc.fr/278591175/id http://www.sudoc.fr/278603947/id
is Praeses of	http://www.sudoc.fr/263051188/id
is Author of	http://www.sudoc.fr/068888716/id Uncoupling of the Hippo and Rho pathways allows megakaryocytes to escape the tetraploid checkpoint TET2-mediated 5-hydroxymethylcytosine induces genetic instability and mutagenesis Mutations in the SRP54 gene cause severe congenital neutropenia as well as Shwachman-Diamond–like syndrome Altered Ca2+ Homeostasis in Red Blood Cells of Polycythemia Vera Patients Following Disturbed Organelle Sorting during Terminal Erythropoiesis https://hal.archives-ouvertes.fr/hal-03447247#id https://hal.archives-ouvertes.fr/hal-04438619#id Impact of interferon on a triple positive polycythemia vera http://hub.abes.fr/edp/periodical/medsci/2024/volume_40/issue_2/msc230306/w A driver role for GABA metabolism in controlling stem and proliferative cell state through GHB production in glioma Different impact of calreticulin mutations on human hematopoiesis in myeloproliferative neoplasms http://hub.abes.fr/edp/periodical/medsci/2015/volume_31/issue_3/medsci20153103p268/w Induced Pluripotent Stem Cells Enable Disease Modeling and Drug Screening in Calreticulin del52 and ins5 Myeloproliferative Neoplasms Stepwise GATA1 and SMC3 mutations alter megakaryocyte differentiation in a Down syndrome leukemia model IFN: Jekyll and Hyde When monoclonal gammopathy‐associated chronic neutrophilic leukemia is a reactive process distinct from a clonal myeloproliferative neoplasm: Lessons from mistakes https://orbi.uliege.be/handle/2268/258335#id Acquired TET2 mutation in one patient with familial platelet disorder with predisposition to AML led to the development of pre-leukaemic clone resulting in T2-ALL and AML-M0 Identification of biallelic germline variants of SRP68 in a sporadic case with severe congenital neutropenia Germline duplication of ATG2B and GSKIP predisposes to familial myeloid malignancies Ph− myeloproliferative neoplasm red blood cells display deregulation of IQGAP1-Rho GTPase signaling depending on CALR/JAK2 status Apoptotic Topoisomerase I-DNA Complexes Induced by Staurosporine-mediated Oxygen Radicals https://hal.archives-ouvertes.fr/hal-02390604#id https://hal.archives-ouvertes.fr/hal-04372481#id Level of RUNX1 activity is critical for leukemic predisposition but not for thrombocytopenia HSP27 is a partner of JAK2-STAT5 and a potential therapeutic target in myelofibrosis HSPCs display within-family homogeneity in differentiation and proliferation despite population heterogeneity Predicting the Long-Term Efficacy of Ifnα in JAK2V617F and Calr-Mutated MPN Patients Inferring the dynamic of mutated hematopoietic stem and progenitor cells induced by IFNα in myeloproliferative neoplasms An inherited gain‐of‐function risk allele in EPOR predisposes to familial JAK2 V617F myeloproliferative neoplasms TET2 haploinsufficiency alters reprogramming into induced pluripotent stem cells Heterozygous and homozygous JAK2(V617F) states modeled by induced pluripotent stem cells from myeloproliferative neoplasm patients P53 activation inhibits all types of hematopoietic progenitors and all stages of megakaryopoiesis. Targeting PP2A-dependent autophagy enhances sensitivity to ruxolitinib in JAK2V617F myeloproliferative neoplasms Mutations in the SRP54 gene cause severe congenital neutropenia as well as Shwachman-Diamond–like syndrome DNA binding of the p21 repressor ZBTB2 is inhibited by cytosine hydroxymethylation Daunorubicin- and Mitoxantrone-Triggered Phosphatidylcholine Hydrolysis: Implication in Drug-Induced Ceramide Generation and Apoptosis Inferring the initiation and development of myeloproliferative neoplasms PPARγ agonists promote the resolution of myelofibrosis in preclinical models Surface-exposed and soluble calreticulin: conflicting biomarkers for cancer prognosis Protein kinase Cζ mediated Raf-1/extracellular-regulated kinase activation by daunorubicin Properties and biological roles of TET proteins during embryogenesis and in hematopoiesis http://www.sudoc.fr/246622792/id Activation des voies anti-apoptotiques par les ligands de mort et les médicaments antitumoraux Analysis of the ten-eleven translocation 2 (TET2) gene in familial myeloproliferative neoplasms. Oxidative stress-induced activation of Lyn recruits sphingomyelinase and is requisite for its stimulation by Ara-C CXCR4/CXCL12 axis counteracts hematopoietic stem cell exhaustion through selective protection against oxidative stress The double-edged sword of adenosine Induced Pluripotent Stem Cells Enable Disease Modeling and Drug Screening in Calreticulin del52 and ins5 Myeloproliferative Neoplasms https://orbi.uliege.be/handle/2268/253020#id https://orbi.uliege.be/handle/2268/255981#id Differential association of calreticulin type 1 and type 2 mutations with myelofibrosis and essential thrombocytemia: relevance for disease evolution. Presence of atypical thrombopoietin receptor (MPL) mutations in triple negative essential thrombocythemia patients. MCM8-and MCM9 Deficiencies Cause Lifelong Increased Hematopoietic DNA Damage Driving p53- Dependent Myeloid Tumors https://hal.archives-ouvertes.fr/hal-04409696#id Defective interaction of mutant calreticulin and SOCE in megakaryocytes from patients with myeloproliferative neoplasms https://orbi.uliege.be/handle/2268/255980#id https://orbi.uliege.be/handle/2268/268268#id https://hal.archives-ouvertes.fr/hal-04438412#id CXCL12/CXCR4 pathway is activated by oncogenic JAK2 in a PI3K-dependent manner Inherited transmission of the CSF3R T618I mutational hotspot in familial chronic neutrophilic leukemia Calreticulin del52 and ins5 knock-in mice recapitulate different myeloproliferative phenotypes observed in patients with MPN CALR mutant protein rescues the response of MPL p.R464G variant associated with CAMT to eltrombopag Disrupted filamin A/αIIbβ3 interaction induces macrothrombocytopenia by increasing RhoA activity Not just another kinase mutation! Germline ATG2B/GSKIP-containing 14q32 duplication predisposes to early clonal hematopoiesis leading to myeloid neoplasms Description of a knock-in mouse model of JAK2V617F MPN emerging from a minority of mutated hematopoietic stem cells Different impact of calreticulin mutations on human hematopoiesis in myeloproliferative neoplasms Lyl-1 regulates primitive macrophages and microglia development

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