About: Emmanuel Brouillet

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An Entity of Type : foaf:Person, within Data Space : data.idref.fr associated with source document(s)

Attributes	Values
type	Person
sameAs	Brouillet, Emmanuel (19..-.... ; neurologue) Brouillet, Emmanuel (19..-.... ; neurologue) Brouillet, Emmanuel (19..-.... ; neurologue)
Life Event	http://www.idref.fr/07891924X/birth
preferred label	Brouillet, Emmanuel (19..-.... ; neurologue)
name	Emmanuel Brouillet
dbpedia-owl:citizenship	http://sws.geonames.org/3017382/
Language	http://lexvo.org/id/iso639-3/fra
Given name	Emmanuel
familyName	Brouillet
has biographical i...tion (Deprecated)	Directeur d'une thèse de : Sciences Biologiques, Université Paris-Sud 11, Orsay (en 2003) Neurologue. Directeur de recherche CNRS, en poste au Laboratoire des Maladies Neurodégénératives (UMR9199, CEA-CNRS-Univ. Paris-Sud, Université Paris Saclay, MIRCen) et à l'INSB Institute of Biological Sciences (UMR 8256, CNRS-Sorbonne Université)
gender	male
ORCID iD	0000-0001-6322-7403
ISI Researcher ID	B-4784-2014
Scopus ID	7004028668
is sameAs of	Brouillet, Emmanuel (19..-.... ; neurologue) Brouillet, Emmanuel (19..-.... ; neurologue)
is Thesis advisor of	http://www.sudoc.fr/228513324/id http://www.sudoc.fr/110675126/id http://www.sudoc.fr/182282058/id http://www.sudoc.fr/155715100/id Etude des mécanismes moléculaires impliqués dans l'infiltration cérébrale des lymphocytes T dans la maladie de Parkinson http://www.sudoc.fr/078908655/id http://www.sudoc.fr/246744383/id Etude de l'amplification de la neurodégénérescence excitotoxique par une dysfonction mitochondriale, implications pour la maladie de Huntington http://www.sudoc.fr/25914861X/id http://www.sudoc.fr/25915234X/id Etude des mécanismes de mort neuronale, in vivo, induits par une atteinte mitochondriale, implication pour la maladie de Huntington http://www.sudoc.fr/246920106/id http://www.sudoc.fr/241692318/id Etude structurale de biomarqueurs de neuropathologies, cas particulier de la protéine CRYM, une Cytosolic-3,3',5-triiodo-L-thyronine(T3)-Binding Protein http://www.sudoc.fr/233068120/id
is Praeses of	http://www.sudoc.fr/190630361/id Modélisation de la dysfonction des neurones dopaminergiques associée à la maladie de Lesch-Nyhan à l'aide des cellules souches induites à la pluripotence http://www.sudoc.fr/194947882/id http://www.sudoc.fr/199207127/id http://www.sudoc.fr/19970127X/id http://www.sudoc.fr/267216734/id http://www.sudoc.fr/277581958/id http://www.sudoc.fr/27301384X/id
is Author of	http://www.sudoc.fr/044049226/id http://www.sudoc.fr/246291311/id 2010001M06rik (St102) protects striatal neurons against an N-terminal fragment of mutant huntingtin in vivo Striatal and nigral pathology in a lentiviral rat model of Machado-Joseph disease Impairment of Glycolysis-Derived l-Serine Production in Astrocytes Contributes to Cognitive Deficits in Alzheimer’s Disease https://serval.unil.ch/notice/serval:BIB_F7B822E6181B#id In vivo benzodiazepine receptor occupancy by CL 218,872 visualized by positron emission tomography in the brain of the living baboon: modulation by GABAergic transmission and relation with anticonvulsant activity Brief Communication Complementarity of gluCEST and $^1$H‐MRS for the study of mouse models of Huntington's disease Silencing ataxin-3 mitigates degeneration in a rat model of Machado-Joseph disease: no role for wild-type ataxin-3? Energy defects in Huntington's disease: Why “in vivo” evidence matters Insulin growth factor-1 protects against excitotoxicity in the rat striatum https://serval.unil.ch/notice/serval:BIB_69446186248E#id The C-Terminal Domain of LRRK2 with the G2019S Substitution Increases Mutant A53T α-Synuclein Toxicity in Dopaminergic Neurons In Vivo Haloperidol protects striatal neurons from dysfunction induced by mutated huntingtin in vivo. Mitochondria in Huntington's disease Discovering new bioactive neuropeptides in the striatum secretome using in vivo microdialysis and versatile proteomics. Complex roles for reactive astrocytes in the triple transgenic mouse model of Alzheimer disease Calpain is a major cell death effector in selective striatal degeneration induced $in\ vivo$ by 3-nitropropionate: implications for Huntington's disease. Modulation of astrocyte reactivity improves functional deficits in mouse models of Alzheimer’s disease https://serval.unil.ch/notice/serval:BIB_18C9D3A78F11#id https://serval.unil.ch/notice/serval:BIB_15371235638A#id ETUDE IN VIVO DE L'INTERACTION DE SUBSTANCES CONVULSIVANTES ET DE SUBSTANCES ANTICONVULSIVANTES AGISSANT AU NIVEAU DU COMPLEXE RECEPTEUR GABA-BENZODIAZEPINE CANAL CHLORE Dopamine determines the vulnerability of striatal neurons to the N-terminal fragment of mutant huntingtin through the regulation of mitochondrial complex II Potentiating tangle formation reduces acute toxicity of soluble tau species in the rat The striatal long noncoding RNA Abhd11os is neuroprotective against an N-terminal fragment of mutant huntingtin in vivo The striatal kinase DCLK3 produces neuroprotection against mutant huntingtin Altered enhancer transcription underlies Huntington’s disease striatal transcriptional signature In vivo expression of polyglutamine-expanded huntingtin by mouse striatal astrocytes impairs glutamate transport: a correlation with Huntington's disease subjects IRC-082451, a novel multitargeting molecule, reduces L-DOPA-induced dyskinesias in MPTP Parkinsonian primates. The Mitochondrial Toxin 3-Nitropropionic Acid Induces Striatal Neurodegeneration via a c-Jun N-Terminal Kinase/c-Jun Module Doublecortin-like kinase 3 (DCLK3), a novel striatum-enriched species, is a modulator of mutant huntingtin in vivo https://serval.unil.ch/notice/serval:BIB_1D85A501F855#id https://serval.unil.ch/notice/serval:BIB_B17434CC555F#id Corticostriatopallidal Neuroprotection by Adenovirus-Mediated Ciliary Neurotrophic Factor Gene Transfer in a Rat Model of Progressive Striatal Degeneration Characterization of the striatal kinase Dclk3 and its neuroprotective effects against mutant huntingtin Age-associated evolution of plasmatic amyloid in mouse lemur primates: relationship with intracellular amyloid deposition https://serval.unil.ch/notice/serval:BIB_A967A49D88F5#id CA150 expression delays striatal cell death in overexpression and knock-in conditions for mutant huntingtin neurotoxicity Neuronal tau species transfer to astrocytes and induce their loss according to tau aggregation state IGF-1 exacerbates the neurotoxicity of the mitochondrial inhibitor 3NP in rats The Neuroprotective Agent CNTF Decreases Neuronal Metabolites in the Rat Striatum: An $in\ Vivo$ Multimodal Magnetic Resonance Imaging Study https://serval.unil.ch/notice/serval:BIB_94F3BC76E274#id Ciliary neurotrophic factor activates astrocytes, redistributes their glutamate transporters GLAST and GLT-1 to raft microdomains, and improves glutamate handling in vivo. Dopamine Gene Therapy for Parkinson’s Disease in a Nonhuman Primate Without Associated Dyskinesia https://serval.unil.ch/notice/serval:BIB_5C3A4B5F5795#id https://serval.unil.ch/notice/serval:BIB_9D3AFCAEBB95#id https://orbi.uliege.be/handle/2268/2909#id https://serval.unil.ch/notice/serval:BIB_C283E4E59574#id Coupling of D2R Short but not D2R Long receptor isoform to the Rho/ROCK signaling pathway renders striatal neurons vulnerable to mutant huntingtin SCA7 Mouse Cerebellar Pathology Reveals Preferential Downregulation of Key Purkinje Cell-Identity Genes and Shared Disease Signature with SCA1 and SCA2 SET translocation is associated with increase in caspase cleaved amyloid precursor protein in CA1 of Alzheimer and Down syndrome patients. https://serval.unil.ch/notice/serval:BIB_258DFC19091E#id Encephalopathy induced by Alzheimer brain inoculation in a non-human primate THY-Tau22 mouse model accumulates more tauopathy at late stage of the disease in response to microglia deactivation through TREM2 deficiency https://serval.unil.ch/notice/serval:BIB_36DDED1148BA#id Imaging and spectroscopic approaches to probe brain energy metabolism dysregulation in neurodegenerative diseases Precision machine learning to understand micro-RNA regulation in neurodegenerative diseases

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