About: Christophe Heymes

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An Entity of Type : foaf:Person, within Data Space : data.idref.fr associated with source document(s)

Attributes	Values
type	Person
sameAs	Heymes, Christophe (19..-....) Heymes, Christophe (19..-....) Heymes, Christophe (19..-....)
Life Event	http://www.idref.fr/174627424/birth
preferred label	Heymes, Christophe (19..-....)
name	Christophe Heymes
dbpedia-owl:citizenship	http://sws.geonames.org/3017382/
Language	http://lexvo.org/id/iso639-3/fra
isni:identifierValid	0000000423364747
Given name	Christophe
familyName	Heymes
has biographical i...tion (Deprecated)	Chargé de recherche à l'INSERM Directeur de thèse, Université Paris 7, 2013 Directeur de recherche membre de l'Institut des Maladies Métaboliques et Cardiovasculaires (I2MC) de l'Université Toulouse 3 Paul Sabatier (UT3) et de l'Institut national de la santé et de la recherche médicale (Inserm) (en 2020)
gender	male
ORCID iD	0000-0002-6666-6193
ISI Researcher ID	Y-9368-2019
Scopus ID	7003599563
is sameAs of	Heymes, Christophe (19..-....) Heymes, Christophe (19..-....)
is Thesis advisor of	http://www.sudoc.fr/174626916/id http://www.sudoc.fr/183374339/id http://www.sudoc.fr/198613709/id http://www.sudoc.fr/24804057X/id Homéostasie des espèces oxygénées réactives et physiopathologie de l'hypertrophie/insuffisance cardiaque, focus sur l'isoforme Nox4 de la NADPH oxydase, comparaison d'un inhibiteur synthétique sélectif GKT137831 avec PROVINOLS tm concentré polyphénolique de vin rouge
is Author of	Structural evidence for a new elaborate 3D-organization of the cardiomyocyte lateral membrane in adult mammalian cardiac tissues ACE inhibition prevents diastolic Ca2+ overload and loss of myofilament Ca2+ sensitivity after myocardial infarction. Red wine polyphenols prevent metabolic and cardiovascular alterations associated with obesity in Zucker fatty rats (Fa/Fa). Cytotoxic CD8+ T cells promote granzyme B-dependent adverse post-ischemic cardiac remodeling Atomic force and electron microscopic-based study of sarcolemmal surface of living cardiomyocytes unveils unexpected mitochondrial shift in heart failure. Liraglutide targets the gut microbiota and the intestinal immune system to regulate insulin secretion Molecular phenomics and metagenomics of hepatic steatosis in non-diabetic obese women A 3‐week nonalcoholic steatohepatitis mouse model shows elafibranor benefits on hepatic inflammation and cell death [Impact of periodontal disease on arterial pressure in diabetic mice]. Neuronal NO synthase mediates plenylephrine induced cardiomyocyte hypertrophy through facilitation of NFAT-dependent transcriptional activity Involvement of β3-Adrenoceptor in Altered β-Adrenergic Response in Senescent Heart: Role of Nitric Oxide Synthase 1–derived Nitric Oxide https://serval.unil.ch/notice/serval:BIB_2602769A64EF#id RhoA activation and interaction with Caveolin-1 are critical for pressure-induced myogenic tone in rat mesenteric resistance arteries The Gut Microbiota Regulates Intestinal CD4 T Cells Expressing RORγt and Controls Metabolic Disease Caveolin-1 and -3 dissociations from caveolae to cytosol in the heart during aging and after myocardial infarction in rat. Carabin Protects Against Cardiac Hypertrophy by Blocking Calcineurin, Ras, and Ca 2+ /Calmodulin-Dependent Protein Kinase II SignalingCLINICAL PERSPECTIVE Ephrin-B1 is a novel specific component of the lateral membrane of the cardiomyocyte and is essential for the stability of cardiac tissue architecture cohesion. Caveolin-1 and -3 dissociations from caveolae to cytosol in the heart during aging and after myocardial infarction in rat Cardiomyocyte overexpression of neuronal nitric oxide synthase delays transition toward heart failure in response to pressure overload by preserving calcium cycling. Cardiac Specific Increase in Aldosterone Production Induces Coronary Dysfunction in Aldosterone Synthase-Transgenic Mice Circulating Microparticles from Patients with Septic Shock Exert Protective Role in Vascular Function Endothelial Dysfunction Caused by Circulating Microparticles from Patients with Metabolic Syndrome Phosphatidylinositol 3-Kinase and Xanthine Oxidase Regulate Nitric Oxide and Reactive Oxygen Species Productions by Apoptotic Lymphocyte Microparticles in Endothelial Cells
is primary topic of	http://www.idref.fr/174627424
is Thesis referee of	http://www.sudoc.fr/185196322/id http://www.sudoc.fr/224090011/id

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